unless we have a pre disposition to taking anything that binds to something that is at the center of many cell activities. This theory would certainly explain the variety of side effects.
a couple of practical but not so scientific points has me thinking along these lines. I took Dut for a month when I was 22 years old to treat an enlarged prostate. I had no issues on or after Dut. So if my issue is in result of having a genetic pre predisposition to experiencing epigenetic changes in result of taking a 5ar inhibitor my gut feeling tells me that this epigenetic change would have occurred immediately at the time of taking Dut how the syndrome manifest’s for everyone else while on or immediately after stopping the drug. Instead, my issues came four years later after taking saw P. That’s when I got PFS. Then three years after that I took Saw P again and got even worse this time completely crushed. So where I am going with this is that it appears for me this was an accumulative thing each time depleting something vital required for optimal functioning and that the difference between me and most PFS victims is that taking Dut once was not enough to deplete what ever this is to give me problems and for me It took “more”. Of course this theory assumes that Dut/Fin work exactly like Saw P does. I have found evidence that supports this and evidence that debunks Saw P working exactly like Fin/Dut. This theory also assumes that an epigentic change cannot occur as an accumulative thing.