Final protocol 100% pfs reversal with dht therapy - lastpost

If that is your main problem Cooper, you should just go live your life.

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Has anyone here got any connection with @JustQuitDut anymore? Is he stilk “cured”? Is he still taking pills to stay cured. I wonder what happened to him and his friend.

I mean no offense, but how badly I wish my problem was “only” low semen volume!

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You’re having a laugh low semen? Man go live, please.

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Update,

I waited for 4 days for my last aromasin dose i hit hyper-sexual libido levels again in 4th day from the last pill.
I took aromasin and today my hyper-sexuality returned to normal, though i can be partially erect if i think about sex without manual stimulation.
Estrogen level is too much tricky to adjust. I will continue to experiment on doses. For now i still have total recovery on libido, visual lust, primary male instinct for sex (even in high stress situations) and genital sensitivity.
My semen is still low but it is very thick and ed is same except more nocturnals and restarted daily random erections.
I am also still using Sulbutiamine and i like it for its mental effects.
It dissipates brain fog a lot but i do not know if, it will have same effects on anyone else.

Guy from this study has also had same effects from arimidex really interesting.

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I also have developed softer erections, my orgasms are not that good anymore, i don’t get morning woods (very rare), and my flaccid penis creates hourglass shape when i have bowel movements. And kinda watery low semen.

The main treatment consisted in Tadalafil 5 mg/day and vacuum therapy, with a favorable but partial response. Being that the diagnostic hypothesis consisted in a drop in the serum levels of DHT, we indicated DHT replacement treatment with Andractim® (2.5% DHT gel) 5 gm per day. Since no clinical response was seen after 3 months of use, it was discontinued. It’s important to clarify that while the dosage was decided according to the treatment of hypogonadism, there is no guideline on the replacement of DHT in patients with this syndrome. After all he had been through, the patient went for a medical consultation at Baylor College of Medicine, Houston, Texas, USA. He wanted to have his DHT levels measured and opt for an alternative treatment. A DHT measurement was performed, as well as a genetic analysis of CAG triplet repeats of the androgen receptor gene, since polymorphisms at this level could be associated to its lower sensitivity to androgens. DHT resulted in a value of 192 pg/ml (112-955 pg/ml), interpreted as a low DHT for his age. CAG determination resulted in 24 repetitions, interpreted as significant for lower sensitivity of the androgen receptor. After this evaluation, the indicated treatment consisted of HCGu 6000 UI/week, divided into 3 applications of 2000 UI/week, combined with Anastrozole 2 mg/week, divided into 2 intakes of 1 mg/week. This was done in order to hyper stimulate the Leydig cells, elevate the levels of Testosterone and blocking the aromatase, thus diverting the metabolism of Testosterone through the 5AR way (assuming it still had any remaining activity), without compromising his spermatogenesis (Figure 3).

After 2 weeks of treatment he noticed some improvements, such as less penile rigidity in a flaccid state, a more “warm” penis, increased morning erections (without any changes in the rigidity of the erections), some growth in penile girth, absence of post ejaculatory asthenia, greater libido, increase in muscular tone and strength, and an improved mood and self-esteem. One month later, a hormonal profile was performed without DHT, showing a Testosterone of 10.2 ng/ml (2.5-8.5 ng/ml), a bioavailable Testosterone of 6.14 ng/ml, and an Estradiol in range, so we decided to lower the dose of HCGu to 5000 UI/week. Two months after this treatment modification, a new hormonal profile was taken, which showed a Testosterone of 7.93 ng/ml, a bioavailable Testosterone of 2.42 ng/ml and an Estradiol in range. This resulted in a more comfortable treatment (given that HCGu is marketed in 5000 UI flasks) as well as a higher sense of well- being. He is currently still under treatment, with a slow but steady improvement in his symptoms, accompanied by periodic evaluation by the Andrology team of the Urology department and the Psichiatry department of the Hospital Italiano from Buenos Aires.

This looks like exactly what happened to me.

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did you take 5000ui of HCG? for how long??

how much time you’re on TRT? in rats testosterone restored mPOA AR levels and sexual behavior in 3 weeks but in humans that would be 1.5 years

Hello brother only difference is that, I do not take any gonadotropins, I only take aromasin Ed fluctuate but, I definitely got improved. I occasionally woke up with bone hard painful boners.

My free T tripled and total T doubled though no change in E2 but E/T ratio is like 1/38.
Also my anxiety is almost zeroed. I am more confident.
My temper from first days of aromatase inhibition is eased a lot.
I try to not to think about Ed just pray for more improvements

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I believe i am in my third month with aromatase inhibition.

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sorry i thought you were on TRT

if you want to follow my second nandrolone trial here it is:

http://www.pssdforum.com/viewtopic.php?f=20&t=3346

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Thank you for the link i have been looking in to that. You mentioned it before and I thought you shared your progress here :).
Well my augmentation plan was to add Dianabol to exemestane but so far I am satisfied with slow but steady improvements.

As a matter of fact we try opposite things let’s see what will happen.

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Do you have Any updates.

I started to get Bone hard Solid erections from out of nothing but, unfortunately it is fluctuating. I suspected from my regular anti histamine usage trying to take a break from it.
Also my nocturnals some times wakes me up in the night i have lost some sensitivity and orgasmic quality but in general every thing is still like pre pfs levels except ED.

Currently i take exemestane every 3rd or 2nd day.

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not yet, it was hard dosing the dutasteride couldn’t get it in solution only suspensions and the dose is in micro range

i forgot if are you on TRT or not?

Not yet on TRT, just taking Aromasin (exemestane) but i have plans to add TRT or switch to tamoxifen.

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Since I increased the aromasin interval, I loose some gains on sensitivity and libido.
Do not misunderstood they are still better than before but they are going down.
This may be because of season changes I do not know, I plan to stop aromasin for two weeks and test what will happen.

interesting, iirc the optimal A/E ratio is 20-50
when i did mine Total T was totally crashed but free T was above the reference range along with e2 which is strange because e2 is supposed to increase shbg

anyway I’ll be done with nandrolone in a couple weeks and do bloods on trt trying to correct this

my IBS improved a lot in the past 2 weeks I’m eating carbs almost everyday without symptoms

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Who is “accompanied by periodic evaluation by the Andrology team of the Urology department and the Psichiatry department of the Hospital Italiano from Buenos Aires”?

I live in Buenos Aires too.

Sorry about dumb questions but english is not my first language.

@mistaker check the link there is information about Doctor you can reach him.

I am state side I do not know why but I got my extreme nocturnals back I quit aromasin 5 days ago I am extremely horny I am not sure about reason may be increasing estrogen or jet lag…

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