Couldn’t agree more.
Yes, we have to approach it this way, otherwise many of us won’t survive long enough to continue the fight. People shouldn’t attack me, I’m trying to keep us all alive and well long enough for something more to happen. That’s what you done understand PVDL, not all of us are willing to go on with our lives and simply “live” with this. I for one am in contact with others and many say the very same thing.
.5mg of arimidex ED is absolute suicide. Literally… In the bodybuilding world we refer to this as s suicide drug. It completely eliminates estrogen from the body. Which can leave far more debilitating effects than what you were probably feeling before. Even in the highest of dosages 1000mg+ of test a week, most used would be .25mg EOD or .5mg E3D. I could not imagine using that dosage on an individual with compromised testosterone levels.
If you are trying to utilize a PCT protocol to bounce back your HPTA, please join my discussion in this forum under (experienced steroid user)
UPDATE, I heard lots of guys are starting the gel plus test, it’s important to keep the gel down to eminem size amounts on each side of your body. Keep the test dose low 10mg a day of prop, if using a long ester, cyp or enanthate, keep it at around 0.4cc of cyp (that’s what goldstein uses) and 0.5cc of enanthate a week.
Note to Dannyfc, do not use masteron yet, first conduct the experiment with the gel and test alone, masteron will shut you down. If the gel doesn’t work with test, I have a protocol to try involving masteron. MCI is on the gel, I am on the gel, RQ is going on the gel, remember, hormones can take up to a month to work, so be patient. We shall see how this experiment plays out, I am only 3 days in, so far haven’t required an AI yet. Still letting dht build up, had a few estrogen surges that lasted minutes and then resolved themselves, was weird, like an inner war. An estrogen surge and then without an AI the dht killed it off. The presence of dht is in fact stimulating 5ar, that theory is correct, and the 5ar is allowing the reduction of test to dht. This is allowing the functions of the 3 5ar enzyme types.
Dr goldstein believes it’s all about the ratio of test to dht, I disagree, it’s lowering neurosteroidal estrogen and raising neurosteroidal dht. The results of such hormonal modifications on a neurosteroidal level result in the ratio altering to what it should be.
My goal for this thread is to make it the best most complete manual for having PFS. If someone gets this thing and wants to read everything in one book, it’s here. They will see how we are, how we feel, my theories, different protocols and responses, and hopefully this thread will end with this protocol now. Imagine if we had this thread to read when we first got this? Combing through this forum desperate and finding one dead end after another, this won’t be the case here.
I wanted to let you guys know that ihatepropecia and I are having some very interesting conversations off of the forum. Both of us are into building muscle and have had the same challenges with hormones and Pfs. We are currently trying to work through them, I may ask him if he wants to bring the conversation into a thread as it would benifit others to hear our discourse.
Do we have enough people for the Baylor studies? Let’s make sure we fill that up, while we are working on solving PFS, we should also make sure to fill up those studies, we have to hit this at every front.
This is a great idea I’ve just set up a recurring payment with paypal, seems a good way to start the new year, we should all be donating something to the PFS Foundation.
is this related to your theory?
eroids.com/forum/steroids-qa … discussion
"
Its a well known myth that steroid
use down regulates androgen
receptors (AR). Steroids actually
do the opposite – they up-regulate
your androgen receptors. There
are more than a few indirect
studies on this (if you care http://
ncbi.nlm.nih.gov/
pubmed/21427060 , Endocrinology
(1990) 126 1165 ect).
The premise of such findings are
that AR are constantly being
turned over. When unattached to
an androgen they have a 1/2 life
of 3 hours and are ultimately
replaced with new ones. But the
presence of an androgen they
become more sensitive, their half
life is doubled and the amount of
new receptors being formed also
increases substantially. Thus
steroid use increases the number
and sensitivity of AR’s.
So if steroids upregulate AR, then
should we be concerned post PCT
when baseline T levels are lower
than before steroid use (when I
mean lower, I mean lower but still
within range). If AR are ‘extra’
sensitive post cycle, then T levels
would be lower than previous
baselines, as less T is needed to
achieve the same baseline effects.
Thats is, pre-steroids you had
levels of 600 (300-1200), but after
using steroids you can only ever
achieve (off-cycle) say 400
(300-1200). In these
circumstances we are not talking
about someone ‘shut down’ just
with lower baseline T levels.
Does this explain why so many
guys who do gear have lower T
levels when they are ‘going
natural’, but feel fine?
"
thats why we have crashed after fin withdrawal - peak androgens - upregulated the ar then all excess androgens aromatized.
second.
Antonio J, Wilson JD,
George FW. Effects of
castration and
androgen treatment
on androgen-receptor
levels in rat skeletal
muscles. J Appl
Physiol
1999;87(6):2016-9.
http://
jap.physiology.org/
content/87/6/2016.
long
The effects of
castration and
dihydrotestosterone
(DHT) treatment on
levels of skeletal
muscle androgen
receptor (AR) were
examined in three
groups of adult male
rats: 1) intact normal
rats, 2) rats castrated
at 16 wk of age, and 3)
rats castrated at 16 wk
of age and given DHT
for 1 wk starting at
week 17. All animals
were killed at 18 wk of
age.
Castration caused a
decrease (P < 0.05) in
the weights of the
levator ani and
bulbocavernosus
muscles. The
administration of DHT
to the castrated rats
increased (P < 0.05)
the weights of the
levator ani and
bulbocavernosus
muscles.
Castration caused a
significant
downregulation of AR
levels in the
bulbocavernosus (P <
0.05) but had no
significant effect on AR
levels in the levator ani
muscle. DHT
administration to the
castrated group
upregulated AR levels
in the bulbocavernosus
and levator ani
muscles. The plantaris
muscle did not
significantly (P > 0.05)
change for any of the
treatments.
These findings
suggest that the
effects of castration
and androgen
replacement
differentially affect
skeletal muscle mass
and AR levels.
Good read but this thread has never been about and is still not about the explanation being releated to the receptors. It’s about estrogen domience and reduced 5 Alpha reductase function.
The best thread about receptors being the problem can be found under theories and was made by someone with the username NYscientist. This Guy claims recovery by taking stuff to fix the receptor problem. Thats his theory anyway.
But JQD is all about estrogen domience and reduced 5AR and or DHT.
whats makes that bullshit exactly? Just curious.
Hey Guys,
Had sex for a couple hours, in total. She cam several times. Wasn’t as hard as last time. Boners between 65%-90%. Drive lower then last time, but noticeable and sex was enjoyable. Last time, I didn’t masterbate for two days prior to sex. Usually I force myself to beat it at least once a day, out of the pathetic hope that something will happen down there. My baseline is near total impotence. Can’t have sex without viagra. So ya, beat it once/twice a day prior to yesterday, so my drive wasn’t as high. All in all, the Adex does the job. The Aromasin also had the same effect, but after a few weeks, boners retreated. JQD speculates this is because Aromasin is chemically similar to DHT, and causes negative feedback (shutsdown testosterone).
Compared to Pre-FIN, I would say Adex brings me to about 30%. My baseline post-FIN is about 1-5%. So it’s a good improvement. Doesn’t compare to pre-FIN, but a big help!
I upped my test to 20mg of propionate today, I had realized 10mg is rather low (from my pre-pfs days), so with this gel I am going to see how things play out, so far so good (but too soon to tell). I inject when I wake up and then I use the gel at night before I go out, I do this so if there is a rise in estrogen from the injection of prop, I have the gel to kill it later on in the night. Something interesting I have noticed, my nails have become much harder than they have ever been, I can’t imagine that is a bad thing.
I have felt 10mg was too little, will let you guys know how it plays out. So basically I wasn’t feeling as great when I lowered my test to 10mg, when my test finally dropped to 10mg, it wasn’t as good, when I upped it to 20mg, I felt great again, so it seems that is my number, but we will see how it plays out. 10mg is very low, especially with PFS, if much of it is being aromatized and the gel is killing the estrogen, we are really only getting like 5mg of test or less even, so that is why I upped it to see how this plays out.
I know Goldstein prescribes 0.4cc of cypionate which is about 140mg of test a week or so, DHT doesn’t do much without enough test.
Homebrew in action. Lol.
Screw Sciene. Long live JQD.
Screw the damn studies, awor and the foundation. Fuck Baylor. Lets invest our money and time in homebrew. For the last 10 years and for the next 10 years. Forever ineffective. Loving it.
Long live 100% PFS REVERSAL WITH DHT THERAPHY.
Do I need to pray at the altar before I rub in the gel?
Hey Tumble, Aromasin doesn’t shut down test, it actually increases it, it’s worse, it shuts down DHT!! I gave it a second try man, only to bloat up like a balloon and have massive estrogen surges within 2 days, taking more just made it worse. It really isn’t something that far off, it is too good to be true that we can take a drug similar to DHT with no consequences, let’s face reality here. It is great for women, but even guys without PFS have told me they start getting estrogen rebound (body builders), one of them has totally quit Aromasin for arimidex.
Don’t over do the arimidex, I would start with testosterone ASAP, if you don’t crash and get all improvements you won’t need the gel, if you crash, you will need the gel. However, you will be able to prevent that with arimidex while you wait for the gel to arrive.
On 20mg of propionate when I wake up and the gel at night before I go out, I feel pretty good, as good as I have ever felt since PFS, will update how things continue to play out, first day I took the gel I was on this dose, seems to be my dose.
[Size=4]"Optional but Highly recommended. Pray to the Divinity you have faith in for full healing intensely on a daily basis! As far as I am concerned this is the most important factor in the entire healing process. But I know full well the hellish anguish and feeling of abandonment by the Divinity and the thoughts of “why me”, “this isn’t fair”, “how could an all-merciful being let this happen?” etc.
So I know perhaps not everyone will do this part, although I pray that you do.
On top of this, if you specifically desire to feel sexually surcharged you should deeply fixate on what previously aroused you the most pre-PFS while you regularly very gently massage the genital area and do aroma therapy with smells that previously excited you especially after taking L-Tyrosine and L-Arginine. BUT DO NOT COME TOO CLOSE TO EJACULATION!" [/size]
For more homebrew ala JQD, visit:
@PLVD, Did you and your brother make it out to Baylor?