If he wants to fix you tell him to follow this:
Exective Summary:
Phase 1: Restore preg, prog and cortisol, via supplementary transdermal pregnenolone (or prog), to as good as can be achieved without thyroid hormones.
Phase 2: Restore thyroid hormones, pref via supplementary slow-release-compounded T3 (not yet T4) and adjust both preg and T3 thyroid hormones together to achieve optimum balance as well as levels of all cortisol-production-line hormones as well as thyroid hormones.
Phase 3: Swap most of the T3 for T4, and swap most of the preg for either dietary cholesterol (pref) or HC (if cholesterol not synthesizing into preg).
Phase 4: Boost GH via GH booster peptides or recombinant GH (if you have the resources to do so).
Phase 5: Boost testosterone and DHT via exogenous T and / or exogenous HCG.
Phase 6: Boost GH if you didn’t boost GH before, but you discovered that your body is tuned for low T / DHT and high GH.
Details:
Phase 1: Restore preg, prog and cortisol, via supplementary transdermal pregnenolone (or prog), to as good as can be achieved without thyroid hormones.
No supplementary thyroid hormones, testosterone, HCG or GH in phase 1.
Transdermal preg is much more reliable at boosting both preg and prog levels, and thus cortisol levels, than oral preg.
Transdermal prog is much more reliable at boosting both prog levels and preg levels too (via “sparing”), and thus cortisol levels, than oral prog.
Most males supplementing with oral preg (or oral prog) will either:
a) synthesize oral preg (or oral prog) into urinary metabolites on the first pass through the liver
or:
b) absorb oral preg (or oral prog) too quickly and cause symptoms of excess preg or excess prog or excess cortisol
Supplementing with plenty of pregnenolone or progesterone is done to ensure at least the first two hormones (preg and prog) in the cortisol-production-line (ie: preg -> prog -> 17 hydroxyprog -> 11 deoxycort -> cortisol) are boosted. Many males discover that transdermal preg synthesizes adequately into cortisol.
The pregneneolone boost will optimize neurotransmitters.
Males react to supplementary transdermal pregnenolone one of two ways:
a) Preg gets backed up (causes “spacey” feelings) with very little cortisol being produced. This is because T4 levels are so low that the adrenal enzymes are “snoozing”.
b) Preg synthesizes adequately into cortisol without preg levels going too high - but cortisol levels go too high even though we don’t feel completely optimized. This spells that the adrenals are adequately invigorated but the rest of our body cells need more T4 in order to absorb more cortisol. Without the T4, a lot of the supplementary cortisol stays in serum (not absorbed into cells).
If your thyroid can produce adequate T4 (younger males only), then this will also boost T metabolism, and you can skip Phase 2 and go straight to Phase 3.
Phase 2: Restore thyroid hormones, pref via supplementary slow-release-compounded T3 (not yet T4) and adjust preg and T3 thyroid hormones to match each other.
No supplementary testosterone or HCG or GH in phase 2.
Compounded-slow-release-T3 spares T4, so the results are more stable than you might assume from T3’s short half life of a few hours.
T3’s short half life is critical to allow you to quickly adjust dosages without having to wait several weeks for T4 serum levels to stabilize.
Supplementing with plenty of thyroid hormone T3 increases metabolic rate some more, and that’s often all that some males need to boost their T metabolism sufficiently for erection performance and orgasms to return to optimum. If this occurs then still go to Phase 3, but probably no further.
When tuning combined preg + T3 doses, you must monitor yawning, tiredness stretching, brain fade, and farting as follows:
…all these can be due to too low thyroid hormones, preventing cortisol from being absorbed into cells (remaining in serum)
…all these can be due to too high thyroid hormones, exhausting cortisol in cells, exhausing pregnenolone, causing neurotransmitter downregulation
…all these can be due to too high cortisol, causing ACTH suppression, causing too low pregnenolone, causing neurotransmitter downregulation
…all these can be due to too low cortisol, due to insufficient pregnenolone
The only way to determine the difference between all of these 4 situations is to have a very good familiarity with the duration that preg and T3 are “effective” in your body, and then determine whether any of your supps has “run out” (last dose of a supp was taken a longer-time-ago than it’s “effective time”), or if your supps are still within their “effective time”, then your most-recently-taken-supplement (preg or T3) is in excess.
Phase 3: Swap most of the T3 for T4, and swap most of the preg for either cholesterol (pref) or HC (if cholesterol not synthesizing into preg)
T4’s half life of several days means you must wait around 2 weeks for serum levels to stabilize before attempting to adjust T4 dosages again. It’s critical that you’ve leaned to tune into your thyroid hormone symptoms from your previous T3 trials, before introducing T4. This helps prevent T4 overdosing. If you overdose on T4, you’ll need to supplement with excess cortisol to force the T4 to be used up (metabolized) quickly.
Phase 4: Boost GH via GH booster peptides or recombinant GH, if you have the resources to do so.
If your T and / or DHT systemic metabolism (eg: libido, but not prostate or hair follicle DHT metabolism) are still too low at this point, boosting GH will spare your T and DHT, because both GH and T trigger many of the same repairs (not 100% overlap) so triggering repairs via GH leaves T to only have to trigger the remaining (fewer) metabolic functions.
Recombinant GH is very expensive in terms of dollars, and travel time to consult the doctor who prescribes it. There are even fewer doctors helping patients dose their GH booster peptides, so while GH booster peptides are lower cost than recombinant GH, you will usually have to travel much further to find a doctor who will work with you to optimize your dosages.
Phase 5: Boost testosterone and DHT via exogenous T and / or exogenous HCG.
This is usually only required by those with insufficient resources to boost their GH in the previous phase.
When some people trial HCG monotherapy, they discover they can reduce their supplementary pregnenolone or cholesterol or HC to ad-hoc only, or even zero.
Phase 6: Boost GH if you didn’t boost GH before, but you discovered that your body is tuned for low T / DHT and high GH
You know your body is tuned for low T / DHT and high GH when supplementing with exogenous T and / or HCG required extremely high cortisol to keep E2 low.
