here i’m,back again…though not joyful as the song sounded.
i even forgot my password of my old account, and failed to retrieve it.
where to begin?
i can’t notice alot of changes on the forum. mew still inquire studies backed by facts,and Boston posted two recoveries,while all of us are pleading for one.
radical opinions like “gene changes” and “androgen receptor insensitivity” still have a place. have you guys seen enough doctors? do you think we are even qualified to treat…let me correct this…do you think YOU are even qualified to treat known medical conditions to go through a complicated situation like this by yourself?
in response to solon…i was prescribed TRT,Nolva and hcg BEFORE being diagnosed of prostatitis. i didn’t even know i have prostatitis. you know it depends on which doctor you visit, if you go to an andrologist he will consider your case from his own perspectives,and hence will manipulate his “tools” trying to fix your case. an andrologist will consider TRT and other modalities he is familiar therewith. an andrologist will not check your prostate if you tell him you have sexual dysfunction.
well,i posted this around 14 months ago.am i right?
i was sent on cipro and hydrocortisone for 2 months,then cipro and hydrocortisone were withdrawn and doxycycline was added for 6 months.still wasn’t enough! the prostate culture turned positive to amoxicillin ,azithromycin and metronidazole. you can’t use amoxicillin for prostatitis, so the dr added azithromycin and metronidazole for 2 weeks,then metronidazole was withdrawn. the culture turned negative to all of those antibiotics except amoxicillin,levofloxacin and ceftriaxone.i was sent on levofloxacin for additional 2 months then quit. why did i quit? because my liver enzymes went high because of all of those drugs. my ALT was 70 and my AST was 55. the doctor said i must stop the drugs.the doctor said that he has never seen such resistant case of prostatitis,at least for a 29 years old man.
the prostate is still tender,large and inflammed. two serum inflammatory markers are high ( ESR and C reactive protein) which means that inflammation didn’t resolve. the etiology of inflammation is unknown.
yeah,this can be an x file. or even worse.
all the possibilities were investigated. first: a decreased immunity. but that was excluded as my WBCs are normal (those were high before starting the antibiotics)
may be an autoimmune prostatitis? but if it was auto-immune,why doesn’t it respond to cortisol anymore?
may be a chronic granulomatous lesion in the prostate…who knows. i just stopped hopelessly.i honestly spent more than enough and my liver was fucked. i will wait for a miracle.
they say that sometimes you have to take a decision that may change your whole life . first time to know that this decision can be swollowing a pill for 60 days so you are in deep shit forever.
i ask everyone on this forum to go and check their inflammatory markers, and to check their prostate. the condition is simply precancerous. chronic unresolved inflammation is simply precancerous.our immune system can’t get rid of that infection.
i’m not telling you myths or radical theories.just go and check your serum inflammatory markers and your prostate. my dr told me that prostate is responsible for erection and libido,and old men who have sexual dysfunction due to prostatitis can’t get better by any TRT. that explains our case.
debunking the nonsense:
1-that finasteride switched off 5-alpha reductase gene: this can’t be true.most of us have normal DHT. even if you have low DHT and started TRT you will have normal DHT again. you can check for 5-a reductase activity by checking its metabolites in urine.
in addition.for a drug to change a gene it must work on DNA.finasteride has no affinity to DNA receptors of testosterone (science,not fiction).finasteride competetively inhibit testosterone binding to the enzyme 5-a reducatse so inhibiting the enzyme from reducing T. fin binds covalently to the enzyme,so the body has to synthesize another enzyme cuz the bond is irreversible.
for a drug to change a gene it must be mutagenic,finasteride was tested for mutagenicity as any drug before clinical trials. it is not mutagenic.
lets face it.we are fucked up for unknown reason! it couldn’t be easier to treat,nor harder. it couldn’t be easier to identify ,nor harder. i recovered in few days and lost my recovery for unknown reason.where does this inflammation come from? this is the million dollar question. if we find an answer,we will hopefully recover before we have forgotten what sex is.
