What i am doing now to try and get back to 100%

Therapeutic Efforts Pharmaceutical
#161

near the tip of the penis, not actually on the shaft, right before the shaft, its like on the bottom rim of the tip, its like these small, little dark redder sores that don’t hurt and aren’t open or anything. i got them checked and my doc was like they can happen from aggressive masturbation or something… but they haven’t really gone away. there’s only two small ones next to each other.

on my middle finger, it’s a bigger scab looking thing, it looks like an open wound was there even though it wasn’t.

although the things on my penis aren’t scab like, they are smaller and not forming scabs, but just the fact that there are sores there plus the wrinkles which was on my middle finger… shows it may be similar to what’s going on.

i talked to someone over PM who was actually a virgin, and he told me he had a staph infection in the past… it’s just way too common that we all have this random thing going on.

#162

FYI

I checked the following website:-
prostatitis.org/prosfaq.html
and it states that ‘Fungal infections of the prostate often reveal themselves by sores which form near the tip of the penis’.

#163

yeah i just read that in the prostatitis thread. it sounds pretty similar and i totally think it’s possible that propecia activated some kind of bad bacteria or it did something, not sure what.

#164

I got the exact same…the zits like bump near the line where hair grows and the sore dark spot on the tip of. My p.

#165

Iwontgiveup - you really have dug up some amazing stuff on that last page. Bacteria/ infection is a difficult road to go down because i don’t think the current medicine even knows half of it. We could all do a bunch of fancy tests but they will never be able to find and treat anything.

As usual, i am simply left wishing that there was some sort of miracle pill that cleaned the body of all infections so that we could rule it out. FUCK!

Good luck to you though mate. Keep it coming.

#166

They won’t find anything because there is nothing TO find.

#167

… :question:

Do you know something we don’t, Mew?

#168

Hi Mew,

You and Awor have alluted to knowing some advances in research… So do you KNOW what it is? If you don’t, then your comment is in fact speculation, but you delivered it as fact.

If you do in fact know something that the rest of us dont, then transparency would be greatly appreciated.

#169

It’s been repeated over and over where the potential problem lies. Nothing has been confirmed 100% yet, but we’re in the right direction. iwontgiveup, I thought you were closest to getting to the problem when you suggested PFS was an aquired case of Kennedy’s disease. PFS is not Kennedy’s disease, but the similarities are too remarkable to ignore. PFS is almost certainly a disease of the AR.

Transperancy isn’t possible at this point. The scientists working on the problem are looking to get their names published, disclosing too much info could jeopardize this, and thus the relationship the PFS community has with the scientists.

The scientists seem to know they are on to something big here, from what I gather. I think this research has a lot of scientific and POLITICAL implications.

#170

No. A disease of the AR cannot possibly explain the high percentage of prostatitis in PFS sufferers, bloody stools that many of us have, improvement with antifungals, worsening with immuno-suppressing corticosteroids such as dexamethasone (me, JG) etc ect. Utter and complete nonsense.

Guess who I’m trying to impersonate here

#171

Is this awor?

And what the similarities, like everyone here has high out of range testosterone?

#172

No, it’s me.

Similarities:

Gynecomastia: breast enlargement.
Impotence
Erectile dysfunction
Reduced fertility
Low sperm count
Testicular atrophy: Testicles become smaller and less functional.
Fasciculations: Twitching of muscles when at rest.
Cramps: Large muscle spasms.
Muscular atrophy: Loss of muscle bulk that occurs when the lower motor neurons do not stimulate the muscle adequately.

Differences: PFS cases seem to respond well to testosterone, but poorly to DHT. Reasons should be obvious.

#173

Please enlighten us

#174

Because our AR’s have become hypersensitive to DHT and not T. I thought you out of all people would know.

#175

So you repeated the “symptom”, where is the explanation? Plus I dont understand how AR rendered hypersensitive to DHT, but not T, makes us more reactive to T vs DHT, did you mean the opposite?

#176

huh?

#177

So this would explain why everyone here has out of range high dht then?

#178

Thank you for editing, and no, I did not repeat any symptoms. I listed them once here and they are available for anyone to see.

The other question is beyond my expertise, but I will give it a shot anyway.

When the DHT came flooding back, the AR gene got down regulated, making the AR unable to bind properly with DHT.

en.wikipedia.org/wiki/Regulation_of_gene_expression

#179

so then i would refer back to the “anonymous” comments posted some time ago from the guy explaining how to reregulate the AR

#180

Second, why not, but another fact is that 5ar is in fact POSITIVELY controlled by DHT and adrogens in general (no negative feedback - the usual thing happening in the HPTA for instance- but the opposite, more DHT actually induces MORE 5ar). This is why some of us (including me, JN, cytochrome…) tryed andractim/proviron to increase DHT. Its did work for JN for a while so I guess he was still sensitive enough to it? Anyway, bottom line, we dont know anything here and any theory is bound to be unscientific speculation based on hints of knowledge, and many of us react so differently to hormones that its impossible to create universal PFS clear-cut laws for everyone.