Too often people on this website are chasing their own tails round and round and round again in circle after circle. Half baked theories are postulated day in day out that are pure guesswork that have absolutely no scientific grounding whatsoever.
The questions abound;
Could it be this theory that has just came into my head, how about another idea- could it be that?
No one seems to want to deal in what is known and what can be done and if you try and deal only in the proven science and dare to call a spade a spade then you are insulted off the site.
Wild speculation equals acceptance on the site, no matter how wild it is, insane or inane- no one insults you for it. Being honest, pragmatic and dealing in the medical reality on the other hand gets you into bother, it poos on peoples dinner, it ruins half baked theories and we can’t have that can we?
How many of these wild theories have forwarded anyone’s situation on this site?
That is a rhetorical question because the answer is none and it will always remain none. This is not an episode of diagnosis murder, you can’t just sit around and work it out.
We have known proven effects of finasteride, we know that it reduces free testosterone via one mechanism or another. If we focus on that and how it should be correctly rectified then we can alleviate and in some cases completely remove the problems induced by finasteride. If more information comes to light that is proven by the medical community we can take that on board and look to see how we can use that proven information to help us further….
But what we cannot do is find answers ourselves to extremely complicated medical endocrinology. No one here is going to discuss potential alterations in androgen receptor response or nureotransmitter alterations etc and suddenly shout eureka!
You are not going to have an idea based on what some guy called Dave said and suddenly have a eureka moment and discover “THE ANSWER”.
You will find no answer at all this way and help no one.
So this type of comment which is typical here (sorry J89 nothing personal just highlighting an attitude on the site).
Is part of the problem not the solution.
I know that the known endocrinology, the known problems and resolving them might not always provide answers for people. But in dealing in scientific/medical reality, I can help improve people’s lot and potentially fix people’s problems, often at least alleviate problems. So it might be mocked or dismissed for not providing all the answers, but one thread where you help someone with pathology that can make a difference is worth a thousand of these threads that help no one and never will.
Engaging in this thread one last time….I’ll leave people to it then.
In this thread, which is it to be?
Are people saying that DHT is the problem or are they saying it is testosterone?
I explain the situation with DHT and say ok well test the theory by having Andractim DHT prescribed, I also say that testosterone IS adversely affected. I make this point because people have been suggesting that it is not a testosterone problem in the long term but a DHT problem.
Then I am told that testosterone is not adversely affected and that it is higher on finasteride.
Now I already know what the situation is both on and off the drug but the point is this;
Where is any of this getting anyone?
So testosterone appears to be fine on finasteride, so what?
Does that mean that the answer is finasteride then?
You see this is rubbish like I said, because it is utterly irrelevant what testosterone is ON the drug given your DHT level on it.
The symptoms tell you that, it doesn’t take a rocket scientist to know that finasteride is not an answer to the very problem it caused.
The finasteride lowered the DHT levels and that left you with terrible symptoms. Then you stop the finasteride and then DHT usually recovers but then testosterone and crucially free testosterone is left low, usually as a result of suppression of the HPTA, sometimes due to elevated SHBG and estradiol sometime not.
The “long term” almost invariably means coming off of finasteride, that is why you are here.
And what does finasteride cause in the long term?
It causes the problems with the hormone that I have already explained and the long term problem tends to be the darn hormone I have stated as in testosterone as opposed to DHT.
You can go back to the theories now and I will leave the thread alone.
P.S
Some people here have problems right under their noses, but sail to far away places in search of answers.
Maybe if a few guys club together and talk about gene expression someone can work out a formulae for androgen receptor alteration on the back of a beer mat for a as yet unknown synthetic medication that could be somehow cooked up that would be favorable in rectifying finasteride based problems. I have a magicians cape with big sleeves if required
