I know guys.
Theres the potential of possible k2 interactions with bacteria or a persons microbiome.
It doesnt mean you might not still need this nutrient, but direct supplementation could cause more problems.
Many bacteria require the same nutrients humans do for growth. The same goes for hormones or hormone depletion.
I gave this example before,
Subclinical atherosclerosis is linked to Dysbiosis via vitamin K2-dependent mechanisms
Vitamin K, Dysbiosis, Atherosclerosis, Cardiovascular disease risk
intestinal decontamination and supplementation with vitamin K2 has the potential to be incorporated into clinical practice as additional preventive measures.
investigate the consequences of vitamin K2 metabolism derangement
In conclusion, patients affected by dysbiosis have higher levels of inactive MGP as well as increased arterial stiffness both of which are early markers for vascular dysfunction. This condition is not influenced by vitamin K2 intake from diet confirming that bacteria are the main source of this vitamin in humans and that vitamin K2 metabolism may be altered as a consequence of small intestinal dysbiosis. Longitudinal studies assessing the role of dysbiosis as a condition that predisposes patients to the development of atherosclerosis are needed; for this category of patients, vitamin K2 supplementation and the treatment of intestinal dysbiosis may be therapeutic alternatives of significant utility.