I have spent hundreds of hours researching over the past few months. I’m not sure why enough people are not looking at this. First, if you don’t know about what dopamine is click on the link at the bottom and read it. It verbalizes exactly what I’m missing.
Also, look at what “Badluck” posted. Below is a part of the article that he posted. Some drugs are known as dopamine antagonists, keeping dopamine from attaching to the receptors.
Some drugs are known as dopamine agonists. These drugs bind to dopamine receptors in place of dopamine and directly stimulate those receptors. Some dopamine agonists are currently used to treat Parkinson’s disease. These drugs can stimulate dopamine receptors even in someone without dopamine neurons.
In contrast to dopamine agonists, dopamine antagonists are drugs that bind but don’t stimulate dopamine receptors.
Antagonists can prevent or reverse the actions of dopamine by keeping dopamine from attaching to receptors.
I believe most of levels of prolactin were raised through a spike in Estrogen and as a result the drug caused dopamine to not bind to the receptors.
Well, it should be easier to get my endo dr to prescribe dopamine, than HGH. If Dopamine does not work, I am still convinced the problem with most of us in the pitutary gland; more specifically high prolactin levels. This problem is absolutely neurological in nature.
courses.washington.edu/conj/bess … inemia.htm
utexas.edu/research/asrec/dopamine.html
kci.org/meth_info/lori/Dopam … nd_You.htm