Progesterone definitely raised my T. It is impossible for me to say why it did this, but my suspicion is that it happened because there is some sort of signaling error, where my body expects a very low amount of DHT. If I block conversion from T to DHT, then my T skyrockets because my body is sensing that DHT has gone down and so produces more T to counteract. My body has the ability to produce T, but for some reason doesn’t think it should.
However, once I quit blocking conversion to DHT and my body senses its presence once again, my T plummetts. I do not view progesterone as any type of solution, which is why I personally chose to discontinue. This is just my line of thinking, and I know there are others on here that share it, however I am by no means claiming it is an absolute truth. I think it’s pretty well documented that our T levels rise considerably while we are on fin, because of this same signaling error. (Our body sees DHT disappear and raises T to counteract)
I also don’t feel qualified to categorize our only alternative solutions as Xyrem or Wellbutrin, nor do they seem like the most promising leads (to me anyway). I took Wellbutrin one day and felt very, very strange, I also didn’t feel it addressed the underlying issues, and I don’t even know enough about GHB (Xyrem) to have an opinion, although there are several threads on here that discuss this. Here is one:
propeciahelp.com/forum/viewtopic.php?t=1286