I haven’t seen this particular article get posted before but find it’s quite revealing - https://www.ncbi.nlm.nih.gov/pubmed/27750143
Particularly the following passages:
In rat models of these conditions, FIN has been shown to block the effects of dopamine receptors in the nucleus accumbens (NAcc), a key terminal of the dopamine mesolimbic system.
And the effects of how FIN changed the following proteins:
Two-dimensional electrophoresis (2-DE) analysis coupled to mass spectrometry revealed significant changes in the expression of nine proteins (CRMP2, PSMD1, STX18, KCNC3, CYP255, GABRP, GABT, PRPS1, CYP2B3),
In researching the above list of proteins ^, these are specifically related to brain growth and axon formation in different, very specific areas of the brain.
If I’m reading this correctly, FIN actually alters the expression of proteins that are specific building blocks of the brain. You can see which aspects of the brain these proteins affect by quickly searching them. I was skeptical about @alteredlife amino acid protocol, but maybe if we give our bodies high levels of essential and non-essential amino acids (ie. proteins), this could help replace the proteins who expression has been altered? Really need a biology whiz to step in here and see if there’s further exploration needed wit these proteins specifically.
Furthermore, they alter the receptors of the nucleus accumbens (NAcc), which according to Wikipedia: “As a whole, the nucleus accumbens has a significant role in the cognitive processing of motivation, aversion, reward (i.e., incentive salience, pleasure, and positive re-enforcement), and reinforcement learning (e.g., Pavlovian-instrumental transfer). In addition, part of the nucleus accumbens core is centrally involved in the induction of slow-wave sleep. The nucleus accumbens plays a lesser role in processing fear (a form of aversion), impulsivity, and the placebo effect.”
This largely reads as a list of all our mental sides (lack of pleasure, motivation, anhedonia, loss of deep-wave sleep/insomnia), and as we know, dopamine and the pleasure centers could have some pretty big implications for libido and our sexual sides as well.
I’m wondering if we increase out dopamine levels, if this might help reset the receptor. I know whenever my dopamine levels are high, my sides completely dissapear and my libido comes back in full force.