More vitamin K2, more testosterone

The list of supplements that boost testosterone production has just got a little longer.
Nutritionists at Tohoku University in Japan say that male rats synthesise more testosterone if given food that contains high amounts of vitamin K2.

ergo-log.com/vitk2testosterone.html

There are two forms of vitamin K: plant-based vitamin K1 [phylloquinone] found in green vegetables such as peas, broccoli and spinach, and the animal-based K2 [menaquinone]. The difference between the two forms is in the ‘tail’ of prenyl units: K2 has one and K1 doesn’t. The tails vary too: meat and eggs have a tail with four extra prenyl units [MK-4], cheese and quark contain various versions of vitamin K2 with seven, eight and nine prenyl units [MK-7, MK-8 and MK-9]. Fermented products like natto contain a K2 vitamin with seven prenyl units.

We need K2 to enable vital enzymes to function. These include enzymes that synthesise coagulant factors and others that fix calcium in the bones – but vitamin K probably has many more functions. The Japanese research was set up to learn more about these unknown functions of vitamin K2.
The researchers gave one group of lab animals food containing low amounts of vitamin MK-4 – 0.75 mg per kg – and the other group got food containing high amounts of vitamin MK-4 – 75 mg per kg. The experiment lasted for five weeks. In the group that got a high amount of MK-4, the animals’ testosterone levels and their testicular testosterone concentration increased as the experiment progressed. The vitamin had no effect on the level of LH, the pituitary hormone that stimulates testosterone production in the testes.

The food with a high vitamin K content caused an increase in the activity of the enzymes PKA and CREB in the testes. And this in turn probably caused the enzyme CYP11A to become more active. This enzyme is responsible for the synthesis of testosterone.

When the researchers repeated their experiment with vitamin K1 they observed hardly any testosterone-boosting effect.

A diet with high levels of vitamin MK-4 “may contribute to the reduced risk of age-related diseases by promoting increased testosterone production in the testis”, the researchers write. Maybe we’ll hear more about this. The Japanese reveal that they have also done experiments with geranylgeraniol, “a side chain structure of MK-4”. This compound and its analogues have been shown to boost testosterone production in cell studies.

I have recently started taking a Vitamin K2 supplement and will update this thread if i notice any significant improvement.

Nice find on the testosterone production aspect of K2.

Be aware that Vit D, K2 and vitamin A need to balance each other, and A can interfere with absorption of D. The retinol form of vitamin A transforms to trans-retinoic acid, the same molecule that the 5 alpha reductase inhibitor Accutane is based upon.

Studies on K2 supplementation in post menopausal women used dosages as high as 45 mg in the form of three divided doses with meals (@ 15 mg); produced no noticeable side effects.
benthamscience.com/cds/samples/cds1-1/Iwamoto.pdf

I take 1 to 4 mg daily, in the form of MK-4 drops on fatty food (butter, egg, bacon etc.) along with vitamin D. No positive effect on Adiol-G nor free testosterone observed in me, from taking this. My free T runs 50 to 140, depending upon clomiphene or topical T doseage, and should be 185 for someone my age.

Kazman. I’ve started taking Life extension Vitamin K2 and some probiotics with some improvement and no side effects.

Do you think its possible that we are suffering from cyp11a enzyme deficiency?
which is the enzyme responsible for the synthesis of testosterone?

Many drugs act as an anticoagulant and antagonize the effects of vitamin K.
Anticoagulants are used in rat poison and work by depleting Vitamin K reserves:

ehow.com/how-does_4673889_rat-poison-work.html

Maybe finasteride has an anticoagulant effect by depleting vitamin K reserves in our bodies?

According to wikipedia:
Mutations in the CYP11A1 gene result in a steroid hormone deficiency, causing a minority of cases of the rare and potentially fatal condition lipoid congenital adrenal hyperplasia:

en.wikipedia.org/wiki/Lipoid_congenital_adrenal_hyperplasia

Problems that emerge in persons with lipoid CAH can be divided into:
1.mineralocorticoid deficiency,
2.glucocorticoid deficiency,
3.sex steroid deficiency, and
4.damage to gonads caused by lipid accumulation.

Good point but if it is just the case of cyp11a enzyme deficiency and low testosterone then would not testosterone replacement fix the issue? or maybe since there is now mutation in cyp11A1? So what is the solution and how will it be diagnosed?

According to the following article:
ncbi.nlm.nih.gov/pubmed/8933406
If you did have cyp11a enzyme deficiency, then you should have certain biochemical abnormalities such as elevated urinary tetrahydro-11-deoxycortisol, elevated serum 11-deoxycortisol, elevated 17-hydroxyprogesterone ,elevated androstenedione, elevated dehydroepiandrosterone sulphate.

There are other enzyme deficiencies that may be relevant to our condition (not just cyp11a).
e.g. 21-hydroxylase deficiency.

Other enzyme deficiencies can be found here:
endotext.org/pediatrics/pediatrics8/pediatrics8.htm

Further info on 21-hydroxylase deficiency including diagnostic tests:
labtestsonline.org/understanding/analytes/6-17hydroxy/tab/faq