Just a wild theory I developed recently but:
the PFS began for many guys weeks after quitting Fin. While there may have been sides while being on Fin, things got really messed up after quitting with very different symptoms than while being on Fin.
5AR1 is responsible for many neurological processes in the brain:
One I mentioned before in another thread is the conversion to Allopregnanalone. A lack of Allopregnanalone e.g. leads to the very common PFS syndrom of insomnia. Now that is just one of many processes where 5AR1 is involved.
In some cases the actions of 5AR1 and 5AR2 are the same, meaning they convert the same precursor to whatever is next in line.
Could it be that the massive surge of 5AR2 after quitting Fin led the body to shut down 5AR1 activity? This could explain many of the neuro sides such as brain fog, insomnia, loss of libido…just to name a few. I am by no means saying that theory is perfect or that it’s the whole picture, but it sounds fairly reasonable in my mind.
Thoughts?