Ok…so these three things really make my dick shrivel up and become really numb/unresponsive to touch. I also have zero libido during this…any thoughts on how the three might be related?..or what is going on that causes this?
To be clear it’s when I fast over a day or during rigorous exercise that this occurs.
I recently did a 2 day fast, and just like you i shriveled up and had no libido during these two days. However I think this is normal/expected. You’re body is depleted of energy during this period.
Exercising the same thing happens, though for me it goes away maybe a half hour after im done with my workout.
Thats weird about the weed use, though. Whenever I smoke weed my dick looks really healthy- like there is normal blood flow down there and nothing is shriveled up.
Generally when doing a fast you don’t see positive results until you break the fast and begin refeeding. If your body thinks you are starving to death, sexual function is not very high on its list of things to divert energy to. Also, fasting for only a day or two isn’t going to accomplish much. If you’re looking for results from a single fast, you need to shoot for a longer time period. Just be very careful and do your research. You can hurt yourself if you fast incorrectly.
Same here with weed, my dick looks great in about a 12 hour peroid after i smoke, erections are great and libido and sensitivity are up.
Exercise…
Well i Its kinda high on my list. Ha!
Well during my high im numb as ever…& after i come down off of it my libido and sensitivity is much higher than usual.
as for exercise the numbness not as bad as when i first got off propecia…i now have alot of energy but it still affects me a little bit.
this could be a far fetched thought but could these activities promote the body to release chemicals (finasteride/estrogen/or whatever) or food that are stored in the body’s fat to sustain the body as an energy souce that this could be causing this? just a thought.
Fasting and exercise increase adrenaline.
Mary Jane increases GABA.
How good is the weed you are smoking?
Weed makes my dick shrivel up and completely numb when it’s flaccid but it increases my libido. A single sexual thought and my sick responds immediately, and I begin to feel that primal sex drive that is foreign since having PFS.
Anyone know about “Rick Simpson Cannabis Oil?”
I’d give it a shot, going on probation soon though…
I have a stash of junk weed and some chronic…I don’t smoke alot though so I haven’t done a thorough comparative analysis
Anybody try eating it? Results?
it all comes down to one’s reaction towards weed. There are also thousands of weed strains that cause different effects in different human beings. I’ve used a vaporizer for two years and it’s a cointoss, sometimes u feel libido (dick is bigger) sometimes its all shriveled up, cold, and zero libido. I think it also has to do on your mental state at that moment, if u are tense/stressed u’ll be more likely to get the shriveled up zero libido -effect.
Oh and I’m sure if you lived in America in a state with medical marijuana laws you could easily qualify for a medical mj use with pfs.
As a general rule though, i think getting high daily reduces your testosterone in the long run and I would advise against it.
A guy on our accutane board tried it and says it cured him 100% of sexual dysfunction, brain fog, etc. It just has to be made very properly using the right strain and rick simpson method. The stuff supposedly can cure a lot of “uncurable” things
which board?
can you buy it somewhere or do you to make it yourself?
Here is the page he posted it on. HIs name is “accutaneispoison” You can’t really buy it anywhere because it is made out of MJ. Simple to make with it though I’ll post a video about it below
I wish someone is going to give this oil a try and report back to us. It seems possible to get more or less the same for medical use in some places (I thinks it’s what they call cannabis tincture). I might see what I can do to get it this way, but I’m not sure some random doctor will let me get in the program and don’t know if the process is long before being accepted.
Cannabis And Multiple Sclerosis.
Over the past twenty years or so, many discoveries concerning the various mechanisms of cannabinoids from the cannabis plant have been unveiled, revealing to an extent how these chemicals can interact with such a broad multitude of diseases.
One particular disease which is becoming increasingly well known for being treatable via cannabinoidal therapeutics is Multiple Sclerosis (Demyeliniation Disease). The term describes an auto-immune disease which effects the nervous system in particular. Inflammation believed to be caused by an immune response, causes the destruction of the myelin sheeth surrounding nervous cells, causing chronic pain and a variety of symptoms.
Western medicine does not offer many solutions to sufferers of the disease. Genuinely, patients experience a whole host of ailments, including chronic tremors, pain, fatigue, swelling, bladder weakness, reduced mobility etc. Under normal circumstances, a singular medication with the capacity to treat all of these symptoms, as well as the underlying cause, would be seen as a modern miracle by the public and medically trained. Unfortunately, the great majority are totally unaware of the potential cannabinoids can have when treating such conditions.
Sources:
Role of cannabinoids in multiple sclerosis.
Although extracts from the cannabis plant have been used medicinally for thousands of years, it is only within the last 2 decades that our understanding of cannabinoid physiology and the provision of evidence for therapeutic benefit of cannabinoids has begun to accumulate. This review provides a background to advances in our understanding of cannabinoid receptors and the endocannabinoid system, and then considers how cannabinoids may help in the management of multiple sclerosis (MS). The relative paucity of treatments for MS-related symptoms has led to experimentation by patients with MS in a number of areas including the use of cannabis extracts. An increasing amount of evidence is now emerging to confirm anecdotal reports of symptomatic improvement, particularly for muscle stiffness and spasms, neuropathic pain and sleep and bladder disturbance, in patients with MS treated with cannabinoids. Trials evaluating a role in treating other symptoms such as tremor and nystagmus have not demonstrated any beneficial effects of cannabinoids. Safety profiles of cannabinoids seem acceptable, although a slow prolonged period of titration improves tolerability. No serious safety concerns have emerged. Methodological issues in trial design and treatment delivery are now being addressed. In addition, recent experimental evidence is beginning to suggest an effect of cannabinoids on more fundamental processes important in MS, with evidence of anti-inflammation, encouragement of REMYELINATION and neuroprotection. Trials are currently under way to test whether cannabinoids may have a longer term role in reducing disability and progression in MS, in addition to symptom amelioration, where indications are being established.
ncbi.nlm.nih.gov/m/pubmed/21323391/
Cannabinoids and multiple sclerosis.
There is a growing amount of evidence to suggest that cannabis and individual cannabinoids may be effective in suppressing certain symptoms of multiple sclerosis and spinal cord injury, including spasticity and pain. Anecdotal evidence is to be found in newspaper reports and also in responses to questionnaires. Clinical evidence comes from trials, albeit with rather small numbers of patients. These trials have shown that cannabis, Delta(9)-tetrahydrocannabinol, and nabilone can produce objective and/or subjective relief from spasticity, pain, tremor, and nocturia in patients with multiple sclerosis (8 trials) or spinal cord injury (1 trial). The clinical evidence is supported by results from experiments with animal models of multiple sclerosis. Some of these experiments, performed with mice with chronic relapsing experimental allergic encephalomyelitis (CREAE), have provided strong evidence that cannabinoid-induced reductions in tremor and spasticity are mediated by cannabinoid receptors, both CB(1) and CB(2). Endocannabinoid concentrations are elevated in the brains and spinal cords of CREAE mice with spasticity, and in line with this observation, spasticity exhibited by CREAE mice can be ameliorated by inhibitors of endocannabinoid membrane transport or enzymic hydrolysis. Research is now needed to establish whether increased endocannabinoid production occurs in multiple sclerosis. Future research should also be directed at obtaining more conclusive evidence about the efficacy of cannabis or individual cannabinoids against the signs and symptoms of these disorders, at devising better modes of administration for cannabinoids and at exploring strategies that maximize separation between the sought-after therapeutic effects and the unwanted effects of these drugs.
Just thought you guys should have a heads up that on our accutane site we now have a SECOND person who has recovered from PFS like symptoms after using Rick Simpson Cannabis Oil.
Here is a link to the page and the guys name is “TaneAbomination”
is there somebody out trying the rick simpson oil? I just saw the documentary ‘‘what if cannabis cured cancer’’ and i think it’s promising enough to give it at least a try. I would, but I don’t have the money for it.
I would love to get my hands on some of this oil. It sounds like the holy grail for our bodies.