I believe It’s more complicated than that. Let’s say the DHT inhibiter caused are prostates for example to have less 5AR activity/expression in them, in other words are prostates now produce less DHT after are abnormal response/crash from a DHT inhibiter.
Well the fact that, that has happened would not reflect in are blood/DHT levels because the DHT can just be getting produced via 5ar activity that is taking place somewhere else in the body. Scientists still don’t even know of every possible place in are bodies 5AR activity takes place in.
But they do know that it deff takes place in the prostate and are entire reproductive systems for that matter. They also know that these are places in the body 5 alpha reductase type 2 inhibitets such as finastride and dutastride target.
Also as justquitdut’s theories explain we need all of the 5AR enzymes that we had prior to are crash to processs, regulate and produce are DHT to testosterone ratios/levels the way are bodies wanted to do on its own But now of course can’t because of missing 5AR enzymes.
So the key is to constantly try to upregulate 5AR activity just as justquitdut is telling everyone to do. Of course this is difficult to do considering the following:
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we have no way to measure 5 alpha reductase activity. (And no the 3abiliog or what ever the hell it’s called is not a way as many members once thought it was)
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we have no drugs that are “clinically proven” to induce 5Ar activity.
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and by the way we need to accomplish this goal of inducing 5Ar while maintaing health total and free testosterone levels the entire time.
Had the mission impossible song started going off in your head yet?
Ohh ya… Forgot to mention you will need to do all of this with out the help of a doctor. Forgot about that part for a sec.
But dont worry we are people who refuse to give up