FKBP51 decreases the activity of DNMT1 and mediates epigenetic effects of the antidepressant paroxetine

“In mouse embryonic fibroblasts and primary mouse astrocytes, FKBP51 mediated several effects of paroxetine, namely, decreased the protein-protein interactions of DNMT1 with CDK5 and FKBP52, reduced phosphorylation of DNMT1, and decreased the methylation and increased the expression of the gene encoding brain-derived neurotrophic factor (Bdnf).”

Could this mean that FKBP51 is important for us (PSSD sufferers specifically)? There exist drugs that target this protein.