I purchased it from nearby pharmacy store, it’s available in my country without Rx.
I have not considered gel but i guess injections can work great.
Update :
Sorry for my long inactivity, I found a lab and started to monitor my blood levels of DHT / E2 Total T. I got %50-%70 percent boost from exemestane on total T my E2 was like 16-18 I switched to more potent Arimidex E2 went under 15 total T increased %90 percent more than 10 ng/ml. DHT was 450 ng/L but after I quit from ai inhibition DHT went down to 205 ng/L ( at the very low end of normal range) E2 increased to 35 pg/ml
With Aromatase inhibitors I had Ed but sensitivity and refraction period was normalized now I lost them.
I have lost more than 30 kg’s of weight I have the best shape ever in my life my cardio capacity is better than triathlon runners my body fat is under 8.5 percent I did keto and low carb now on carnivore diet all off these had little impact on my ED… only things that works are aromatase inhibitors for pleasure and desire and pde5 inhibitors for ED…
Hmmm
First time I have ever heard this with decease in estradiol levels from AI increasing sensitivity and refectory period but making ED worse. Maybe if Era or Erb are down regulated reduction in estrogen levels makes estrogen receptors more sensitive resulting in increased sensitivity and refractory. But why would Estrogen receptors go out of wack again immediately upon stopping AI?
Like everything else with PFS your experience is not consistent across the bored. AI makes all my sexual sides substantially worse. Have you ever tried cycling AI’s. I mean it they don’t make you worse and clearly target something that temporarily recovers some of your your sexual sides maybe cycling them to see if doing so results in a permanent state of equilibrium
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I would just go on TRT + Masteron if i were responding like u. Risking such potent drugs like arimidex is a time bomb.
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Armidex made my sexual sides worse years ago
Armidex seems very risky. But then there are people who say it makes them Better. Wish we could find a way to know which category we will fall in before we trial it
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No i don’t believe pfs has anything to do with estrogen, then why shall i take it !! And if i have elevated e2 it must be due to low dht, i just need proviron or masteron for that. Breast cancer medications like arimidex is not good for men unless they want osteoporosis.
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I think that mine had something to do with estrogen
Why else would have temporarily inhibiting aromatase enzyme with armidex made my sexual sides worse
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Becoz estrogen is as important as androgens, inhibiting e2 can cause severe weakness of bones, will destroy peristalsis of stomach, fucks up brain function. E2 is very important for men and some people here consider e2 as their sole enemy and claim on arimidex which fucks up people with normal e2.
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I understand
But that still does not change the fact that many steroid guys take Armidex for the short term while on steroid cycles and don’t get a shrunken dick, reduced sensitivity, poor orgasm and reduced semen quality. Certainly Most don’t don’t continue to suffer from these sides permanently after discontinuing the Armidex
After already having PFS I took Armidex and it worsened all these sexual sides I already had
Clearly not a normal non PFS Guys Response to short term Armidex usage
Inhibiting aromatase enzyme already in a PFS state clearly touched on a part of the underlying mechanism of my PFS disorder. Not on a level of Just “generally speaking Armidex is bad for guys” .
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There are many people in this forum who has got pfs (or post aromatase inhibitor syndrome) from arimidex like drugs. They never ever took fin. @Jamie @gents93 @lowe2sucks just to name a few. Arimidex fucks with estrogen receptors just like fin does with androgen ones.
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Yes true . I’m familiar with post AI syndrome
I think the estrogen receptors Erb and Era play a role in PFS.
There is a connection with serotonin and these receptors. I’ll post info on that when I have a chance. And I’m currently on the neurotransmitter path . So I believe there is a connection. Especially seeing that when I increase serotonin my constipation is cured.
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How do you supposedly ‘increase serotonin’?
Here is what I know:
Fact number (1) : I took a urine neurotransmitter test and seen that the amount of serotonin in my urine was not in the optimal range
Fact number (2) : I took L-Tryptophan, 5-HTP and vitamin b6 for 64 days which are all things involved in the synthesis of serotonin
Fact number (3) : I took another urine neurotransmitter test and seen that my amount of serotonin in my urine was now in the optimal range
Fact number (4): my constipation was cured
Fact number (5): without doing any additional testing beyond the before and after testing in the initial experiment I came on and off L-Tryptophan, 5-HTP and b6 five times and every time the constipation returned. Every time I went back on the L-Tryptophan, 5-HTP and b6 my constipation was cured again
Fact number (6): I don’t know what the serotonin levels in my CNS are
Fact number (7): Anything NOT outlined in facts 1-6 related to serotonin levels in my CNS or any other assumptions that cannot be reasonably inferred by any of these facts are NOT things that I am presenting as facts
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Yes it is dangerous, I wouldn’t try it if I knew how some people are destroyed from it yet, it improved me so much I returned from castrated state to somehow normal state… in my latest ultrasound I discovered that, I developed some calcification in my prostate my PSA is almost zero so, seems no BHP involved. I will do cycling but i am afraid to use it until the point of no return but, almost every time when I start to use it, I absolutely got restored sensitivity and normalized refractory period with no clear help on Ed.
What did you do again ? Increase serotonin?
I Did nothing I quit from everything and waited… as expected libido went down refractory period increased… on rutin exam they found my prostate stones…
It seems finally I have found something, i have normal total T (6.02 ng/ml) but I discovered that I have low bioavailable T holding at 1.82 ng/ml
Free T 0.252 nmol/ml (under lower limit which is 0.255)
Bioavailable T %30.3 percent…
I think aromasin fixes this so I felt some restoration on libido and desire…
I got Doppler results it seems I have venous insufficiency… how the hell aromatase inhibitor helps on vascular issue …
If you are located in the US I really think you’d be better off just going to a TRT clinic.
UG stuff you never know what you’re getting.
Also be really careful with Anastrozole. I know you don’t have used Finasteride but all the data we have show that PFS have very low estradiol in the central nervous system. 20 folds lower than controls.
Estradiol is very protective of the brain. So it might not be the best idea to use AIs at this point. Without estrogens in the brain dopaminergic neurons die and that’s not reversible.
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Hey @Cbrandel, I haven’t heard of this estrogen theory before about the 20 folds lower in PFS victims. Do you have any sources you could share?
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