This was discussed heavily last year and this article didn’t break new ground. I don’t have energy to go back over every thing that’s double posted, I’m not sure about the rest. You can’t cherry pick hormones that JN took and single them out as the cause for his recovery. In the previous threads we discussed iodine and found that some of us, per the skin test, were deficient. However if you have a RT3 problem Iodine will only fuel it, you need to take only T3, as both T3 and RT3 are derived from T4 which the body gets one way or another as a result of armour or Iodine.
I tried to treat with Iodine and it briefly caused improvements before putting myself in a worse position. The problem is our bodies don’t respond as they should to these treatments due to how propecia physiologically altered our bodies. Even if metabolism speeds up it needs a lot more than just T3/Cortisol to sustain.
With Propecia we shut off DHT + a cortisol pathway + dihydropregnalone or dyhydroprogesterone (can’t remember which) while simultaneously spiking testosterone and estrogen. These are all key hormones for the body’s proper neurological and sexual function. Some of them occur in the brain and have further downstream effects. Simply replacing cortisol or T3 is not going to cure the whole problem IMO. It is also possible that finasteride literally shut off or killed off areas of the brain, or pathways in the brain, that we need for sexual function.
Nevertheless if you choose to go down this path I’m sure we’d all like to see updates. I’m not trying to be too much of a downer but don’t assume your article isn’t read just because the concept is new to you.
Very, very interesting and it may be a clue as to how some of us are predisposed to propecia problems. I personally believe - from my low body temperature, dry skin, hairloss etc that i have thyroid problems that now have come to light since propecia has sent my adrenals up the left. It’s no coincidence at all that many guys have mounted decent improvements (And recoveries) from things like dexamethasone and T3 and also, in JN’s case preg and T3. I think the above is a vital clue for us, imo. I’m currently working on a few things in this regard and will report back in the coming months.
I’m definately taking into consideration what he’s saying about “going natural” to address thryoid problems as opposed to synthetic T3 etc; iodine is particularly interesting if one presents with only a mild thyroid problem.