he is definetly onto something…why don’t all these doctors that have some experience with this colloborate together and come up with treatments?
"Unless your dihydrotestosterone (DHT) level is already very low, you may be wasting your time and money unless you precede the DHT treatment phase with a DHT “starvation” phase by using a 5-alpha reductase inhibitor such as finasteride (Proscar® and Propecia®) to partially block the conversion of testosterone to DHT and thereby increase your subsequent sensitivity to DHT. The primary reason why penis size stagnates in adult men is not because they don’t have DHT (most already have plenty), but because their androgen receptors are functionally desensitized to it (by “functionally desensitized,” I mean the lack of sensitivity could lie with the receptor or its interaction with DNA). Once you rebound from the DHT starvation phase (i.e., once you discontinue the 5-alpha reductase inhibitor), you may not even need supplemental DHT. This isn’t theoretical speculation—I’ve done it and can speak from experience (see my “DHT diary” earlier in the book). You may wish to use DHT:
►To prolong the penile growth phase that accompanies the DHT rebound phase (my rebound phase lasted about three weeks). In my case, I would not have begun using supplemental DHT until the fourth week. Prior to that time, my endogenous DHT was sufficient.
►To increase your response in the early DHT rebound phase if your endogenous DHT isn’t adequate.
As a rule when using supplemental hormones, use the least amount that produces the desired effect. People differ in their response to hormones, so I cannot tell you that you would need “x” number of milligrams per day to achieve a certain effect, whether that effect is increasing penis size or bench pressing 300 pounds. You might think that “more is better” and “it couldn’t hurt” in the case of deciding whether to use supplemental DHT in the early DHT rebound phase. I do not agree. If you’ve read the analogies I used in this book, you know that hormones do not produce effects by themselves; they are just signals that certain effects are needed. Remember my foreman/factory analogy in which the foreman represented the hormone and the factory represented the cellular machinery that produced the proteins? Keep that in mind and consider this: if the factory is already working at full speed, it does not do any good for the foreman to shout, “Work faster!” If your penile cells are growing at maximal speed, adding more DHT will not accelerate their growth and may even hurt it long-term by reducing receptor sensitivity. Hence, it pays to be judicious in deciding whether you should use DHT and, if so, determining its dose and timing. One thing is clear: your receptor sensitivity will decrease in time (mine did after three wondrous weeks). At that time, you can use supplemental DHT with less concern over its impact on receptor sensitivity. Had I used supplemental DHT, here is how I would have done it:
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Week #1: No supplemental DHT.
Week #2: No supplemental DHT.
Week #3: No supplemental DHT.
Week #4: Use supplemental DHT.
Week #5: Use more supplemental DHT than I used last week.
Week #6: Use even more supplemental DHT than I used last week.
In advance, I would not know how much supplemental DHT to use. I would determine the correct amount not by thinking of it in terms of milligrams, but in terms of assessing its response. I already had a good baseline during those first three weeks for judging what maximal (I think) penile growth was like. In weeks #4 through 6 (or even longer), I would use just enough DHT to maintain growth. I would not try to maintain growth at the phenomenal rate I experienced the first three weeks. Why not? Receptor sensitivity375 is bound to decline, and if you use enough DHT in an attempt to maintain the initial rate of growth, you may reduce receptor sensitivity even more. If you err on the side of using too little DHT you may grow at a slower speed, but you will likely retain the capacity to grow for a longer time."