Sigh…
You guys can BELIEVE whatever you want, but that DOESN’T MAKE IT TRUE.
If you want to take someone’s drive-by word who simply posts without any research to backup their claims, be my guest because you are really putting yourself at risk. Did you even read this guy’s prior posts? Just a rambling mess, in broken english, based on one person’s personal opinion with NO SCIENTIFIC RESEARCH used to bolster his “theories”.
And this is exactly what he does – and probably gets satisfaction out of doing – coming on here every few months/years, stirring shit up again with the same old spiel, getting people to argue, and start the whole process anew with uninformed users thinking “well, maybe he’s onto something…”.
In fact, at one point in his history, he had a qualified medical diagnosis COMPLETELY unrelated to any of his proposals, which had to do with a liver issue: propeciahelp.com/forum/viewt … sc&start=0 .
So go ahead, if you “believe” this theory:
Tell me how Chlamydia pneumoniae has anything to do with penile shrinkage, peyronie’s disease and fibrotic tissue changes which can occur while on the drug due to depleted DHT, and when post-Fin Testosterone levels drop?
Tell me how Chlamydia pneumoniae has anything to do with Erectile Dysfunction, a documented side effect of the drug, which has been linked to loss of DHT and nitric oxide activity in penile tissues thanks to Finasteride?
Tell me how Chlamydia pneumoniae has anything to do with Gynecomastia, which is a listed side effect of the drug due to Finasteride’s documented alterations of the androgen/estrogen ratio in favor of estrogen?
Tell me how Chlamydia pneumoniae has anything to do with muscle atrophy, which is a direct result of loss of androgen levels/possible androgenic action at the androgen receptor?
Tell me how Chlamydia pneumoniae has anything to do with reduced fertility and semen volume, both direct consequences of Finasteride’s mechanism of action?
Tell me how Chlamydia pneumoniae has anything to do with loss of spontaneous, nocturnal and particulary morning erections – documented in the Propecia Clinical Trials submitted to the FDA – which are a sign of declining Free Testosterone levels in men?
Tell me how Chlamydia pneumoniae has anything to do with complete loss of libido which is a direct result from the loss of androgen levels/function post-drug when so many men on this site find their T levels crash to hypogonadal levels?
Tell me how Chlamydia pneumoniae has anything to do with depression and anxiety, two undocumented side effects by Merck but which are reported in numerous scientific published journals via Finasteride’s inhibition of 5AR-reduced neurosteroids Allopregnanolone & THDOC?
I could go on and on – the point is, Candida, Chlamydia pneumoniae and Chlamydia trachomatis are NOT RESPONSIBLE for ANY of these effects. They have been discussed on here in the past – just because you even test positive for chalmydia pneumoniae antibodies does not even mean it is a cause, as many people come into contact with this bacteria throughout their lifetime in some form or another. This quackery has already been debated and debunked numerous times on this forum in the past, and on numerous other websites. For example, the user’s original theory of Candida as the source of problems due to intestinal “toxins” – quackwatch.com/01QuackeryRel … ndida.html
Again, if people want to grasp for straws and believe anything, they will be no further ahead. If you want to believe in SCIENCE, then review the Finasteride Studies section and you’ll start to understand what the drug actually does, as a starting point for investigation. Otherwise, you could claim your problems are from any infinite number of things, Finasteride-related or not.
Please, think logically. Anyone can post anything on the Internet and make claims – very few can successfully backup their points with scientifically, peer-reviewed PUBLISHED RESEARCH. Until there’s a paper written on the connection between Finasteride and Chlamydia, this theory belongs in the trash bin as far as I’m concerned. It is a complete red herring and will only steer people down the wrong path, wasting both their time, energy, money and potentially, expose them to damaging effects from unnecessary use of powerful antibiotics. That is MY personal opinion, I don’t claim to have all the answers, but based on all the research done to date, and my and others’ conversations with scientific researchers, most paths seem to point to 5AR2 enzyme activity, androgen metabolism and the androgen receptor as areas of focus.
The fact I continue to give these posts the time of day is actually an error on my part. In the future they will be deleted if he posts them again, to stop this endless cycle of nonsense.