As I said I do not believe in that androgen independent theorie. It should be: Despite androgen (partially) deprivation, c.cells grow.
So, whether they are extremely over expressed in order to run “properly” as those cells want to survive. C. is somehow a survival program. Anything that disturbs the normal cellular metabolism will cause that program in order to survive.
Now to the theory: I read whole the theory several months ago and I agree in most of the points. What we don’t know if everybody on fin has AR overexpression. We need to test this!
If so, what cag repeats does he have? I guess the longer ones. = ppl with higher androgen levels due to a greater insensitivity to androgens.
When this ppl get androgen deprivation, what happens to their lower sensitivity? Correct, they get AR upregulated in order to get androgen pathway running. That is the goal.
Test doesn’t decrease on fin, it even gets higher. But E2 gets higher as well. So, you get a puffy face, feminine face. E2 is trying to inhibit fin. Here we we are.
You have upregulated AR. I even guess you had bigger muscles and big energy while on fin, but also weight, gain, right?
I want to study your case a bit more. That’s why it would be interesting to have some other parameters.
I will come back here after reading your posts.
Cheers